By Brian L. Samuels, John E. Ultmann (auth.), John E. Ultmann, Brian L. Samuels (eds.)

ISBN-10: 1489972943

ISBN-13: 9781489972941

ISBN-10: 1489973052

ISBN-13: 9781489973054

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While clear-cut morphological and immunohistological differences between Ki-1 + ALC lymphoma and HD have not yet been found, the comparison of chromosomal aberrations observed in Ki-1 + ALC lymphoma and HD disclosed differences supporting the view that these two tumors are separate entities. Although 150 years have passed since Thomas Hodgkin described the disease that later bore his name, several questions remain unanswered, or have not been answered to everybody's satisfaction: (1) What is the cellular origin of Hodgkin and Reed-Sternberg cells?

These data indicate that the tumor Table 5. Detection of EBV-specific DNA by PCR in lymph node biopsies. Hodgkin's disease Ki-P ALC lymphomas Non-Hodgkin's lymphomas B-CLL Normal lymph nodes 114/198 6/23 21/128 1/20 2/20 (57%) (26%) (16%) (5%) (10%) 36 cells in HD are associated with EBV in a high percentage of cases. This frequent association between EBV and HD fits with other serological and epidemiological studies [26] showing an increased risk of developing HD in patients with previous infectious mononucleosis and high titers of IgG antibodies to viral capsid antigen and other viral structures.

Br J Cancer 1989; 60: 83-88. 13. Moore SG, Gooding CA, Brasch RG et a!. Bone marrow in children with acute lymphocytic leukaemia: MR relaxation times. Radiology 1986; 160: 237-240. 14. Thomsen C, Sorensen PG, Karle H et al. Prolonged bone marrow T 1-relaxation in acute leukaemia. In vivo tissue characterisation by magnetic resonance imaging. Mag Reson Imag 1987; 5:251-257. 15. Roberts N, Smith SR, Edwards RHT. Characterisation of bone marrow disorders using quantitative magnetic resonance imaging and image analysis techniques.

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Annals of Oncology: Official Journal of the European Society for Medical Oncology by Brian L. Samuels, John E. Ultmann (auth.), John E. Ultmann, Brian L. Samuels (eds.)


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